Flubromazolam peptide is an unusual manmade sedative substance of the benzodiazepine class which is extremely effective (active in the microgram range) with an unusually long 18 hour duration. When administered, it produces anxiolytic, disinhibiting, sedative, muscle relaxant, and memory suppressing effects.
Flubromazolam peptide is renowned for its potential use in the interim treatment of anxiety, insomnia, acute seizures, and the sedation of hospitalized patients. However, it is currently used as a recreational psychoactive substance and exclusively sold by online research chemical vendors as it has not been formally considered.
Only speculations arise with regards to its pharmacology based upon the subjective effects it prompts and its structural similarity to triazolam, pyrazolam and other benzodiazepines.
It is already known that the sudden cessation of benzodiazepines can be potentially dangerous or lethal for individuals using it steadily for long periods of time, sometimes resulting in seizures or death. So it is very advisable to check one’s dose by slowly reducing the daily consumption for a prolonged period of time instead of stopping immediately.
Flubromazolam Peptide Review
Flubromazolam peptide is classified among benzodiazepines. Its name is derived from the fluorine, bromine, and triazole substitutions on its core benzodiazepine framework (FLUorine-BROMine-AZOLe-AM). This chemical also contains a 1, 4 diazepine ring bonded to a substituted benzene ring. Bromine is bound to this bicyclic structure at R7. Additionally, a fluorine replaced phenyl ring is fused to this structure at R5.
Flubromazolam also contains a methylated triazole ring bonded to and integrating R1 and R2 of its diazepine ring. Distinguished by the suffix “-zolam”, flubromazolam belongs to a class of benzodiazepines containing this fused triazole ring, called triazolobenzodiazepines.
Flubromazolam Peptide Reveiw
Benzodiazepines bind to the receptor site and magnifying the efficiency and effects of the neurotransmitter gamma aminobutyric acid (GABA) by acting on its receptors. This produces multiple effects. As this site is the most prolific inhibitory receptor set within the brain, its modulation results in the sedating effects of flubromazepam on the nervous system.
Binding to voltage-dependent sodium channels rather than benzodiazepine receptors may be responsible for the anticonvulsant properties of benzodiazepines.